TRIGEMINAL NEURALGIA
Electric shocks in the face, seconds long, triggered by touching your cheek or brushing your teeth. It is one of the few conditions in medicine where the description alone is close to diagnostic.
What it is and what it is not
Trigeminal neuralgia is brief, paroxysmal, electric-shock-like pain in one or more divisions of the trigeminal nerve, provoked by innocuous stimuli. The attacks last seconds to under two minutes, there is usually a refractory period afterward, and between attacks most people are pain-free.
Constant burning facial pain is a different entity. Facial pain that lasts hours is a different entity. Bilateral facial pain in a young person raises multiple sclerosis and needs that excluded. Getting the classification right is the whole of the treatment decision, which is why the European Academy of Neurology guideline devotes as much attention to it as it does.
Why it appears on a neck website
Two reasons, both practical. First, the trigeminal system and the upper cervical roots converge on the same brainstem nucleus, which is why upper cervical pathology can produce facial pain and why facial pain patients are sometimes sent to us. Second, people with facial pain accumulate a long list of negative investigations and arrive looking for anybody who will work the differential properly.
We are not the definitive service for classical trigeminal neuralgia. We are frequently the service that establishes it is trigeminal neuralgia rather than a cervical referral, a cervicogenic headache, a dental problem or a temporomandibular disorder — and that distinction is where most of the wasted years go.
The workup that should happen
- MRI, and specifically sequences that visualize the trigeminal root entry zone, because neurovascular compression and secondary causes both need identifying.
- Neurological examination — sensory loss in the trigeminal territory points away from the classical form and toward a secondary cause.
- A dental assessment where it has not been done, because dental pathology mimics this and irreversible dental work performed on trigeminal neuralgia is a recognized and avoidable harm.
- A cervical examination, which is the part that is usually missing.
Treatment, stated plainly
Carbamazepine and oxcarbazepine are first-line medical treatment and the response to them is characteristically good — good enough that a complete lack of response is itself a reason to revisit the diagnosis. Where medication fails or is not tolerated, microvascular decompression and the ablative and radiosurgical options exist and have a real evidence base.
Our role sits before that: confirming the diagnosis, addressing any cervical or myofascial component that is adding to it, and making sure the referral that follows is the right one rather than the first one. We do not route anybody straight to a surgical opinion without working the diagnosis first, and facial pain is the area where that policy earns its keep most often.
The overlap nobody mentions
A person can have classical trigeminal neuralgia and a myofascial or upper cervical pain problem at the same time. When the shocks are controlled on medication and a background ache remains, that residual is frequently the second problem — and it is treatable. Attributing it to incomplete control of the neuralgia leads to dose escalation that does nothing.
The trigger map is worth writing down
Note exactly what sets it off and where the trigger zone is — a specific patch of cheek, a tooth, the gum, the corner of the lip, cold air, chewing, shaving, brushing. That map identifies the affected division and is more useful diagnostically than most of what gets ordered.
It also helps practically. People stop eating and stop brushing on the affected side, and the resulting weight loss and dental decline become their own problems on top of the pain.
Why the diagnosis gets delayed
The first stop for facial pain is usually dental, because that is where facial pain sensibly starts. When examination and imaging of the teeth are normal, the path forward is often unclear, and people cycle between services while the attacks continue.
The description of the pain — brief, electric, triggered by light touch, with pain-free intervals — is close to diagnostic on its own. It is asked for less often than it should be, and it is the first thing we ask about.
Secondary causes, and why imaging is not negotiable
Trigeminal neuralgia can be classical, idiopathic, or secondary to an identifiable lesion — multiple sclerosis and posterior fossa tumors being the ones that must not be missed. The classification systems separate these deliberately, and the separation drives everything that follows.
Sensory loss on examination, bilateral symptoms, onset under forty, or any additional neurological sign all shift the probability toward a secondary cause. Those are the findings that turn imaging from good practice into an urgent requirement.
Living with it while the diagnosis is being sorted out
Practical measures matter more here than in most pain conditions because the triggers are the activities of daily life. A soft diet during a bad period. Lukewarm rather than cold water. A scarf across the face in wind. An electric razor. Chewing on the unaffected side.
None of that treats the condition, and all of it makes the interval until treatment works survivable. Weight loss and dental neglect during an uncontrolled period cause real secondary harm and are worth heading off deliberately.
What people ask about facial pain
Is it a brain tumor?
That is what most people are afraid to ask. Secondary causes exist, which is exactly why imaging is part of the workup rather than optional, and the large majority of scans do not show one. The occipital nerve block.
Could it be my teeth?
It could, and it also frequently is not — which is why irreversible dental work should wait until the diagnosis is settled. Extractions performed on undiagnosed trigeminal neuralgia are a well-documented problem. TMJ, or the neck?.
Why do the shocks come from touching my face?
Light touch triggering severe pain is the hallmark of the condition and it is a genuine feature of how the nerve is behaving, not sensitivity or exaggeration. How a cervicogenic headache is identified. Brief electrical pain is a nerve firing, and the trigger is diagnostic.
Will I need surgery?
Many people are controlled on medication for years. Surgery is for medication failure or intolerance, and it is a decision taken with a secure diagnosis in hand. The occipital nerve block.
Related reading
- TMJ, or the neck?
- Cervicogenic headache
- Occipital nerve block
- Temporal arteritis
- CRPS
- Sternocleidomastoid spasm
Have facial pain worked up before anything irreversible happens
The most common preventable harm in facial pain is treatment delivered to the wrong structure.
12174 Natural Bridge Rd, Suite 302
St. Louis, MO 63044
Next to DePaul Hospital, just off the 270 and 70 junction, west of the airport.
Sources
- Bendtsen L et al. European Academy of Neurology guideline on trigeminal neuralgia. Eur J Neurol, 2019. PubMed 30860637
- Bendtsen L et al. Advances in diagnosis, classification, pathophysiology, and management of trigeminal neuralgia. Lancet Neurol, 2020. PubMed 32822636
- Lambru et al. Trigeminal neuralgia: a practical guide. Pract Neurol, 2021. PubMed 34108244
- Ashina et al. Trigeminal neuralgia. Nat Rev Dis Primers, 2024. PubMed 38816415
- Araya et al. Trigeminal Neuralgia: Basic and Clinical Aspects. Curr Neuropharmacol, 2020. PubMed 31608834
